What is Neuropathic Pain?
Neuropathic pain can also be referred to as Neuralgia. It is described as the sensations of;
- Stabbing Pains
- Cold or Hot Sensations
- Electric Shocks
- Pins & Needles
Is it Nociceptive or Neuropathic Pain?
This is caused by damage to tissues. For example, a cut, a burn, an injury, pressure or force from outside the body, or pressure from inside the body (for example, from a tumour) can all cause nociceptive pain. The reason we feel pain in these situations is because tiny nerve endings become activated or damaged by the injury, and this sends pain messages to the brain via nerves.
This type of pain is caused by a problem with one or more nerves themselves. The function of the nerve is affected in a way that it sends pain messages to the brain. In simple terms, neuropathic pain can occur without a correlating issue, whereas nociceptive pain occurs due to a current issue your body needs to address. (Patient.info, 2017)
When it comes to treating patients with either disc bulges, disc herniations or neuropathic pain in general, their symptoms are the initial target which show improvements. This specifically relates to the level of pain the patient is suffering from.
HOW DO STEM CELLS WORK TO TREAT THESE CONDITIONS?
The SVF cells have the potential to;
- Target and fight off inflammation
- Repair damaged nerve cells
- Differentiate to the matching tissue type (in this scenario: nerve cells)
- Soften scar tissue formation (Condé-Green et al., 2016) (Wu et al., 2013) (Gibbs et al., 2015) (Jo et al., 2014) (Michalek, 2015) (Sacerdote et al., 2013) (Vickers et al., 2014)
Inflammation is the common attraction for these cells. The chronic inflammation has not been resolved by your immune system. These cells are able to target and resolve the inflammation around the nerves which will restore normal function. Any nerve cells which are damaged have the potential to be repaired; however nerve cells which are completely dead and not functional will not be inflamed therefore they will not attract the cells and will not be replaced by them.
This has been confirmed with patient studies of Trigeminal Neuralgia.
(Vickers et al., 2014)